The involvement of epithelial-to-mesenchymal transition in doxorubicin resistance: Possible molecular targets
Abstract
Considering the fact that cancer cells can switch among various molecular pathways and mechanisms to ensure their progression, chemotherapy is no longer effective enough in cancer therapy. As an anti-tumor agent, doxorubicin (DOX) is derived from Streptomyces peucetius and can induce cytotoxicity by binding to topoisomerase enzymes to suppress DNA replication, leading to apoptosis and cell cycle arrest. However, efficacy of DOX in suppressing...
Paper Details
Title
The involvement of epithelial-to-mesenchymal transition in doxorubicin resistance: Possible molecular targets
Published Date
Oct 1, 2021
Volume
908
Pages
174344 - 174344
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