Original paper
Differing effects of denosumab and alendronate on cortical and trabecular bone
Abstract
Vertebral fractures and trabecular bone loss are hallmarks of osteoporosis. However, 80% of fractures are non-vertebral and 70% of all bone loss is cortical and is produced by intracortical remodeling. The resulting cortical porosity increases bone fragility exponentially. Denosumab, a fully human anti-RANKL antibody, reduces the rate of bone remodeling more than alendronate. The aim of this study was to quantify the effects of denosumab and...
Paper Details
Title
Differing effects of denosumab and alendronate on cortical and trabecular bone
Published Date
Feb 1, 2014
Journal
Volume
59
Pages
173 - 179
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Notes
History