C2orf69 mutations disrupt mitochondrial function and cause a multisystem human disorder with recurring autoinflammation
Abstract
BACKGROUND. Deciphering the function of the many genes previously classified as uncharacterized open reading frame (ORF) would complete our understanding of a cell’s function and its...
Paper Details
Title
C2orf69 mutations disrupt mitochondrial function and cause a multisystem human disorder with recurring autoinflammation
Published Date
Jun 15, 2021
Volume
131
Issue
12
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