CPT1A-mediated Fat Oxidation, Mechanisms, and Therapeutic Potential
Abstract
Energy homeostasis during fasting or prolonged exercise depends on mitochondrial fatty acid oxidation (FAO). This pathway is crucial in many tissues with high energy demand and its disruption results in inborn FAO deficiencies. More than 15 FAO genetic defects have been currently described, and pathological variants described in circumpolar populations provide insights into its critical role in metabolism. The use of fatty acids as energy...
Paper Details
Title
CPT1A-mediated Fat Oxidation, Mechanisms, and Therapeutic Potential
Published Date
Jan 4, 2020
Journal
Volume
161
Issue
2
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Notes
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