Original paper
Transcriptional Signatures of Cellular Plasticity in Mice Lacking the α1 Subunit of GABAAReceptors
Abstract
GABA A receptors mediate the majority of inhibitory neurotransmission in the CNS. Genetic deletion of the α1 subunit of GABA A receptors results in a loss of α1-mediated fast inhibitory currents and a marked reduction in density of GABA A receptors. A grossly normal phenotype of α1-deficient mice suggests the presence of neuronal adaptation to these drastic changes at the GABA synapse. We used cDNA microarrays to identify transcriptional...
Paper Details
Title
Transcriptional Signatures of Cellular Plasticity in Mice Lacking the α1 Subunit of GABAAReceptors
Published Date
May 24, 2006
Journal
Volume
26
Issue
21
Pages
5673 - 5683
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Notes
History