Syndecan-1 shedding by meprin β impairs keratinocyte adhesion and differentiation in hyperkeratosis
Abstract
Dysregulation of proteolytic enzymes has huge impact on epidermal homeostasis, which can result in severe pathological conditions such as fibrosis or Netherton syndrome. The metalloprotease meprin β was found to be upregulated in hyperproliferative skin diseases. AP-1 transcription factor complex has been reported to induce Mep1b expression. Since AP-1 and its subunit fos-related antigen 2 (fra-2) are associated with the onset and progression of...
Paper Details
Title
Syndecan-1 shedding by meprin β impairs keratinocyte adhesion and differentiation in hyperkeratosis
Published Date
Aug 1, 2021
Journal
Volume
102
Pages
37 - 69
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