MiR‐128‐3p alleviatesTNBS‐induced colitis through inactivatingTRAF6/NF‐κBsignaling pathway in rats

miR‐128‐3p is reported to involve in pathogenesis of several autoimmune diseases, yet the role of miR‐128‐3p in inflammatory bowel disease (IBD) remains unknown. To investigate miR‐128‐3p in IBD, experimental colitis animal model was generated by 2,4,6‐Trinitrobenzenesulfonic acid solution (TNBS). miR‐128‐3p agomir was used to overexpress miR‐128‐3p in rats. Histological assessment and myeloperoxidase activity were conducted to evaluate the...