Blockade of mini-TrpRS for treatment of diabetic foot syndrome
Abstract
Diabetic foot syndrome demonstrates wound chronicity due to impaired tissue perfusion in lower limbs. Previous studies showed interferon-gamma (IFN-γ), a central inflammatory mediator in diabetic foot syndrome, to induce the truncated form of tryptophanyl-tRNA synthetase (mini-TrpRS) that has strong angiostatic properties. Recently we reported that mini-TrpRS signalling could be blocked in the presence of IFN-γ with D-tryptophan in vitro. Here...
Paper Details
Title
Blockade of mini-TrpRS for treatment of diabetic foot syndrome
Published Date
Jan 1, 2020
Journal
Volume
6
Issue
6
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