Parenterally Delivered Methionyl-Methionine Dipeptide During Pregnancy Enhances Mammogenesis and Lactation Performance Over Free Methionine by Activating PI3K-AKT Signaling in Methionine-Deficient Mice

Volume: 150, Issue: 5, Pages: 1186 - 1195
Published: May 1, 2020
Abstract
Pregnancy-induced hypoaminoacidemia, l-methionine (Met) included, disturbs embryogenesis and may also affect breast function. Supplementation with the dipeptide l-methionyl-Met (Met-Met) may improve lactation performance.We compared the effects of supplemental Met or Met-Met during pregnancy on mammogenesis and lactogenesis and investigated underlying mechanisms.In experiment 1, 9-wk-old ICR mice (n = 72, ∼30 g) were divided into 3 groups....
Paper Details
Title
Parenterally Delivered Methionyl-Methionine Dipeptide During Pregnancy Enhances Mammogenesis and Lactation Performance Over Free Methionine by Activating PI3K-AKT Signaling in Methionine-Deficient Mice
Published Date
May 1, 2020
Volume
150
Issue
5
Pages
1186 - 1195
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