Hepcidin is an endogenous protective factor for osteoporosis by reducing iron levels
Abstract
Postmenopausal osteoporosis is a global health issue. Although a lack of estrogen is considered the major reason for postmenopausal osteoporosis, other factors might also contribute the etiology of the disease. In previous reports, we and others proposed that iron accumulation after menopause accelerates osteoporosis, and here, we genetically modified the expression of an endogenous hormone, hepcidin, to modulate iron status in a mouse model....
Paper Details
Title
Hepcidin is an endogenous protective factor for osteoporosis by reducing iron levels
Published Date
May 1, 2018
Volume
60
Issue
4
Pages
299 - 308
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