The IDO-AhR Axis Controls Th17/Treg Immunity in a Pulmonary Model of Fungal Infection.

Volume: 8, Pages: 880 - 880
Published: Jul 24, 2017
Abstract
In infectious diseases, the enzyme indolemine 2,3 dioxygenase (IDO) that catalyzes the tryptophan (Trp) degradation along the kynurenines (Kyn) pathway has two main functions, the control of pathogen growth by reducing available Trp and immune regulation mediated by the Kyn-mediated expansion of regulatory T cells via Aryl hydrocarbon receptor (AhR). In pulmonary paracoccidioidomycosis caused by the dimorphic fungus Paracoccidioides...
Paper Details
Title
The IDO-AhR Axis Controls Th17/Treg Immunity in a Pulmonary Model of Fungal Infection.
Published Date
Jul 24, 2017
Volume
8
Pages
880 - 880
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