Mechanisms of Acetaminophen Hepatotoxicity: Do We Need JNK for Cell Death?
Abstract
During the last decade, c-jun N-terminal kinase (Jnk) emerged as central player in the pathophysiology of acetaminophen (APAP)-induced liver injury, especially in mice.1Han D. et al.Trends Pharmacol Sci. 2013; 34: 243-253Abstract Full Text Full Text PDF PubMed Scopus (134) Google Scholar, 2Du K. et al.Expert Opin Drug Metab Toxicol. 2015; 11: 1769-1779Crossref PubMed Scopus (49) Google Scholar Jnk activation and translocation of...
Paper Details
Title
Mechanisms of Acetaminophen Hepatotoxicity: Do We Need JNK for Cell Death?
Published Date
Aug 1, 2016
Journal
Volume
151
Issue
2
Pages
371 - 372
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