Bee Venom Accelerates Wound Healing in Diabetic Mice by Suppressing Activating Transcription Factor‐3 (ATF‐3) and Inducible Nitric Oxide Synthase (iNOS)‐Mediated Oxidative Stress and Recruiting Bone Marrow‐Derived Endothelial Progenitor Cells

Multiple mechanisms contribute to impaired diabetic wound healing including impaired neovascularization and deficient endothelial progenitor cell (EPC) recruitment. Bee venom (BV) has been used as an anti‐inflammatory agent for the treatment of several diseases. Nevertheless, the effect of BV on the healing of diabetic wounds has not been studied. Therefore, in this study, we investigated the impact of BV on diabetic wound closure in a type I...