Blood pressure rise following angiogenesis inhibition by bevacizumab. A crucial role for microcirculation
Abstract
Arterial hypertension (HT) has been reported in all studies involving bevacizumab, an antiangiogenic agent designed to target vascular endothelial growth factor (VEGF). The mechanism underlying bevacizumab-related HT is not yet clearly understood. As far as endothelial dysfunction and microvascular rarefaction are hallmarks in all forms of HT, we tested the hypothesis that anti-VEGF therapy could alter the microcirculation in nontumor tissues...
Paper Details
Title
Blood pressure rise following angiogenesis inhibition by bevacizumab. A crucial role for microcirculation
Published Date
May 1, 2008
Journal
Volume
19
Issue
5
Pages
927 - 934
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