Advanced glycation endproduct induces ROS accumulation, apoptosis, MAP kinase activation and nuclear O-GlcNAcylation in human cardiac myocytes.

Volume: 80, Issue: 11, Pages: 1051 - 1056
Published: Feb 20, 2007
Abstract
null null Accumulation of advanced glycation endproduct (AGE) has been implicated in the pathogenesis of diabetic complications. However, the precise role and mechanism behind AGE-associated diabetic heart injury are not fully clear. This study was designed to evaluate the effect of AGE on accumulation of reactive oxygen species (ROS), apoptosis, mitogen-activated protein kinase (MAPK) activation and nuclear null O -GlcNAcylation in fetal human...
Paper Details
Title
Advanced glycation endproduct induces ROS accumulation, apoptosis, MAP kinase activation and nuclear O-GlcNAcylation in human cardiac myocytes.
Published Date
Feb 20, 2007
Volume
80
Issue
11
Pages
1051 - 1056
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