Caveolin-3 Inhibits Cav3.2 (α1H) Currents and Regulates Hypertrophic Signaling in Ventricular Myocytes
Abstract
Voltage gated T-type Ca2+ channel Cav3.2 subunit, responsible for T-type Ca2+ current (ICa,T) is expressed in different tissues including the heart and participates in Ca2+ influx, hormonal secretion, pacemaker activity and arrhythmia. The Cav3.2 channels are reported to be up regulated and contribute to the altered Ca2+ signaling and pathogenesis of cardiac hypertrophy via the activation of calcineurin/nuclear factor of activated T cells (NFAT)...
Paper Details
Title
Caveolin-3 Inhibits Cav3.2 (α1H) Currents and Regulates Hypertrophic Signaling in Ventricular Myocytes
Published Date
Feb 1, 2011
Journal
Volume
100
Issue
3
Pages
528a - 528a
Citation AnalysisPro
You’ll need to upgrade your plan to Pro
Looking to understand the true influence of a researcher’s work across journals & affiliations?
- Scinapse’s Top 10 Citation Journals & Affiliations graph reveals the quality and authenticity of citations received by a paper.
- Discover whether citations have been inflated due to self-citations, or if citations include institutional bias.
Notes
History