Koyal Jain
University of North Carolina at Chapel Hill
Internal medicineSurgeryAutoantibodyIntensive care medicineAdverse effectImmunologyMultidisciplinary approachBiopsyVasculitisHemolytic anemiaAntiphospholipid syndromeVascular diseaseThrombotic microangiopathyJaundiceDiseaseThrombocytopenic purpuraLupus nephritisQuality of life (healthcare)AbdomenPsychological stressKidneyCreatinineAcute kidney injuryAscitesDifferential diagnosisPathogenesisNephropathyPopulationExisting TreatmentANCA-Associated VasculitisVery high riskAnca vasculitisDiabetic kidneyRenal survivalFamily medicineMedical emergencyNephrologyDiabetes mellitusMedicineGastroenterology
6Publications
4H-index
21Citations
Publications 10
Newest
#1Samantha L Gelfand (Brigham and Women's Hospital)
#2Koyal Jain (UNC: University of North Carolina at Chapel Hill)H-Index: 4
Last. Amanda K. Leonberg-Yoo (UPenn: University of Pennsylvania)H-Index: 8
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#1Jill R. Krissberg (Stanford University)H-Index: 1
#4Yi Cai (Boston Children's Hospital)H-Index: 1
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#1Koyal Jain (UNC: University of North Carolina at Chapel Hill)H-Index: 4
#2Pankaj Jawa (UNC: University of North Carolina at Chapel Hill)H-Index: 1
Last. Ronald J. Falk (UNC: University of North Carolina at Chapel Hill)H-Index: 86
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Anti-neutrophil cytoplasmic autoantibody (ANCA) vasculitis is a small vessel vasculitis (SVV) resulting in inflammation of small- and medium-sized blood vessels. Since the initial description of SVV, there have been tremendous advances in our understanding of the pathogenesis. Over the last decade, we have made significant progress in understanding the pathogenesis and improving the treatment and prognosis of patients with ANCA vasculitis. Patient and renal survival has improved, and treatment i...
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#1Elizabeth S. Kotzen (UNC: University of North Carolina at Chapel Hill)H-Index: 4
#2Sanjeet Roy (UNC: University of North Carolina at Chapel Hill)H-Index: 2
Last. Koyal Jain (UNC: University of North Carolina at Chapel Hill)H-Index: 4
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Antiphospholipid syndrome (APS) and other causes of thrombotic microangiopathy (TMA) negatively impact the renal outcomes of patients with systemic lupus erythematosus (SLE) and lupus nephritis. Here we review the diagnosis and management of occlusive renal vascular lesions due to APS and other TMAs, with a focus on patients with SLE and lupus nephritis. The presence of a thrombotic event, unexplained hypertension, thrombocytopenia, or hemolytic anemia should prompt consideration for TMA syndrom...
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#2R. J. Falk (UNC: University of North Carolina at Chapel Hill)H-Index: 16
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#6Michelle Hladunewich (Sunnybrook Health Sciences Centre)H-Index: 33
#7Jonathan J. Hogan (UPenn: University of Pennsylvania)H-Index: 4
Rationale & Objectives Glomerular diseases, including minimal change disease, focal segmental glomerulosclerosis, membranous nephropathy, and immunoglobulin A (IgA) nephropathy, share clinical presentations, yet result from multiple biological mechanisms. Challenges to identifying underlying mechanisms, biomarkers, and new therapies include the rarity of each diagnosis and slow progression, often requiring decades to measure the effectiveness of interventions to prevent end-stage kidney disease ...
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#1Koyal Jain (UNC: University of North Carolina at Chapel Hill)H-Index: 4
#2Amy K. Mottl (UNC: University of North Carolina at Chapel Hill)H-Index: 16
IN BRIEF Diabetic kidney disease carries a heavy burden, both economically and in terms of quality of life, largely because of its very high risk for vascular disease. Coordinated, multidisciplinary care with attention to appropriate, timely screening and preventive management is crucial to reducing the morbidity and mortality of this devastating disease.
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#1Koyal Jain (UNC: University of North Carolina at Chapel Hill)H-Index: 4
#2Akanksha Gupta (UNC: University of North Carolina at Chapel Hill)H-Index: 1
Last. Abhijit V. Kshirsagar (UNC: University of North Carolina at Chapel Hill)H-Index: 36
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A 55-year-old man with a history of colorectal cancer, status-post wedge resection of liver 3 weeks prior, was admitted with jaundice, ascites, and acute kidney injury. On presentation, he was icteric with a soft distended abdomen and mild ascites. Laboratory data revealed a creatinine level of 2.72 mg/dl (baseline 1 mg/dl). Total and direct bilirubin levels on admission were 42.5 mg/dl (up from 1.6 mg/dl) and 25 mg/dl, respectively. Numerous dark casts were seen in the urine (Figure 1, red arro...
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#1Pankaj JawaH-Index: 1
#2Koyal JainH-Index: 4
Last. Shiang-Cheng KungH-Index: 1
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#1Koyal JainH-Index: 4
Last. Andre A. Kaplan (UConn: University of Connecticut)H-Index: 22
view all 4 authors...
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