Yusuke Matsuo
Tokyo Medical and Dental University
Internal medicineRadiologyGenetically modified mouseCross-sectional studyPathologyVimentinAntibodyMonoclonalExternal validityChemistryMesenchymal stem cellCohort studyImmunologyBiopsyAzathioprineArthritisTocilizumabParabiosisHealth assessmentHyperplasiaRheumatoid arthritisSynovial membraneFemoral veinMediterranean dietDiseaseMEDLINEBone marrowFibroblastFibrosisPathogenesisBehcet's diseaseImmune complex glomerulonephritisProliferation MarkerLineage tracingImmunoglobulin g4In patientJoint damageWall thicknessEarly raDisease activityCancer researchClinical researchMedicineGastroenterology
7Publications
2H-index
24Citations
Publications 6
Newest
#1Tetsuro Aita (Fukushima Medical University)H-Index: 1
#2Yusuke MatsuoH-Index: 2
Last. Takashi Yoshioka (Fukushima Medical University)H-Index: 5
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#1Yusuke Matsuo (Tokyo Medical and Dental University)H-Index: 2
Last. Takashi Yoshioka (Fukushima Medical University)H-Index: 5
view all 6 authors...
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#1Yusuke Matsuo (Tokyo Medical and Dental University)H-Index: 2
#2Tetsuya Saito (Tokyo Medical and Dental University)H-Index: 4
Last. Hitoshi Kohsaka (Tokyo Medical and Dental University)H-Index: 33
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Fibroblasts play crucial roles in the pathogenesis of rheumatoid arthritis (RA). Accumulation of fibroblasts in the synovial tissues characterizes the pathology of RA. Understanding how fibroblasts accumulate could lead to discovery of new therapeutic targets in RA treatment, while current antirheumatic therapies still have problems in efficacy and safety. In this regard, several studies have revealed cellular origins of fibroblasts in fibrotic tissues in murine models of organ fibrosis. Some st...
1 CitationsSource
#1Yusuke Matsuo (Tokyo Medical and Dental University)H-Index: 2
#2Fumitaka Mizoguchi (Tokyo Medical and Dental University)H-Index: 14
Last. Hitoshi Kohsaka (Tokyo Medical and Dental University)H-Index: 33
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Synovial fibroblasts play crucial roles in inflammation and joint destruction in rheumatoid arthritis (RA). How they accumulate in the RA joints remains unclear. This study was conducted to discern whether cellular influx from the outside of the joints and local proliferation are responsible for synovial fibroblast accumulation in an animal model of RA. We found that synovial fibroblasts were identified as GFP+ cells using collagen type I alpha 2 (Col1a2)-GFP transgenic reporter mice. Then, bone...
9 CitationsSource
#1Yusuke Matsuo (Tokyo Medical and Dental University)H-Index: 2
#2Fumitaka Mizoguchi (Tokyo Medical and Dental University)H-Index: 14
Last. Nobuyuki Miyasaka (Tokyo Medical and Dental University)H-Index: 78
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14 CitationsSource